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Evidence from recent years has indicated that ferroptosis participates in many heart diseases, including myocardial infarction (8), cardiac ischemia/reperfusion (I/R) injury (9), heart failure (10), myocardial hypertrophy (11), sepsis (12), and doxorubicin-induced heart injury (13)

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AHK-Cu significantly stimulates vascular endothelial growth factor (VEGF), a key driver of angiogenesis, or the formation of new capillaries that deliver oxygen and nutrients to dermal tissue