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To enhance neuronal GSH concentrations, several studies have evaluated the chelation of synaptically released zinc using a thiol-containing chemical compound, N-acetyl-cysteine (NAC)

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Consistent with these in vivo effects, we found that bradykinin-stimulated production of NO was enhanced by vector-mediated GPx1 overexpression in cultured endothelial cells at baseline and in the presence of exogenous homocysteine [60]